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0 reviewsAbstract Cerebral edema is characterized by fuid accuand MRI. SalB/Rg1 inhibited matrix metalloproteinase-9 mulation, and the glymphatic system (GS) plays a pivotal (MMP-9) activity, reduced cleaved β-dystroglycan (β-DG), role in regulating fuid transport. Using the Tenecteplase sysand stabilized aquaporin-4 (AQP4) polarity, which was veritem, magnesium salt of salvianolic acid B/ginsenoside Rg1 fed by colocalization with CD31. Our fndings indicated (SalB/Rg1) was injected intravenously into mice 4.5 h after that SalB/Rg1 treatment enhances GS function and attenumiddle cerebral artery occlusion and once every 24 h for ates cerebral edema, accompanying the regulation of the the following 72 h. GS function was assessed by Evans blue MMP9/β-DG/AQP4 pathway.imaging, near-infrared fuorescence region II (NIR-II) imaging, and magnetic resonance imaging (MRI). SalB/Rg1 had Keywords Cerebral edema · Glymphatic system · signifcant efects on reducing the infarct volume and hemIschemic stroke · SalB/Rg1 · MMP9 · AQP4orrhagic transformation score, improving neurobehavioral function, and protecting tissue structure, especially inhibiting cerebral edema. Meanwhile, the infux/efux drainage of IntroductionGS was enhanced by SalB/Rg1 according to NIR-II imaging