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(Ebook) Lung Biology in Health and Disease Volume 177 Therapeutic Targets in Airway Inflammation 1st Edition by N Tony Eissa, David P Huston ISBN 082470956X 9780824709563

  • SKU: EBN-1903394
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Authors:N. Tony Eissa, David P. Huston
Pages:1000 pages.
Year:2003
Editon:1
Language:english
File Size:7.29 MB
Format:pdf
ISBNS:9780824709563, 9780824755171, 082470956X, 0824755170
Categories: Ebooks

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(Ebook) Lung Biology in Health and Disease Volume 177 Therapeutic Targets in Airway Inflammation 1st Edition by N Tony Eissa, David P Huston ISBN 082470956X 9780824709563

(Ebook) Lung Biology in Health and Disease Volume 177 Therapeutic Targets in Airway Inflammation 1st Edition by N Tony Eissa, David P Huston - Ebook PDF Instant Download/Delivery: 082470956X, 9780824709563
Full download (Ebook) Lung Biology in Health and Disease Volume 177 Therapeutic Targets in Airway Inflammation 1st Edition after payment

Product details:

ISBN 10: 082470956X 
ISBN 13: 9780824709563
Author: N Tony Eissa, David P Huston

This reference examines the cellular, molecular, and genetic mechanisms involved in airway inflammation, as well as the pathophysiology, epidemiology, and aetiology of asthma. It explores strategies to prevent cellular injury and oxidative tissue damage, inhibit key inflammatory pathways and identify disease-specific targets to reduce the induction

(Ebook) Lung Biology in Health and Disease Volume 177 Therapeutic Targets in Airway Inflammation 1st Table of contents:

1 Pathophysiology of Airway Inflammation in Asthma
2 Primary Human Lung Cells in the Evaluation of Airway Inflammation
3 Airway pH Homeostasis in Asthma and Other Inflammatory Lung Diseases
4 Monitoring of Exhaled Breath to Assess Airway Inflammation in Asthma
5 The Modulatory Effects of Carbon Monoxide on the Inflammatory Response
6 Reactive Nitrogen Species and Tyrosine Nitration in Airway Inflammation
7 S-Nitrosothiols in Lung Inflammation
8 Contribution of Nitric Oxide Synthase Isoforms to Allergen-Induced Airway Hyperresponsivenessand Inflammation
9 Molecular Mechanisms of Increased Nitric Oxide in Airway Inflammation
10 Regulation of Nitric Oxide Synthesis by Inducible Nitric Oxide Synthase
11 Novel Strategies for Selective Inhibition of Inducible Nitric Oxide Synthase
12 Leukocyte Recruitment in Allergic Pulmonary Inflammation
13 Neutrophil Emigration in the Lungs
14 Dendritic Cells
15 Roles of Mast Cells in Airway Hyperreactivity, Inflammation and Remodeling,and Immunoregulation in Asthma
16 Mast Cell Exocytosis in Airway Inflammation
17 Mast Cell Proteases
18 Peroxidases and Asthma
19 Chemokines in Allergic Airway Inflammation
20 Role of the Eotaxin Subfamily of Chemokines in Allergic Eosinophilic Inflammation
21 Interleukin-4 and Interleukin-13 in Human and Experimental Asthma
22 Interleukin-5 and Eosinophils as Therapeutic Targets for the Treatment of Asthma
23 Modulating the Interleukin-5 Response in Asthma
24 The Role of Interleukin-9 in Allergic Inflammation of Asthma
25 Epithelial Production of IL-12 p80 During Viral Infection and Asthma Supports an Altered Paradigmfor Airway Inflammation
26 Interleukin-16 and Airway Hyperresponsiveness in Asthma
27 Modulation of the Allergic Response
28 Inhibition of Cytokine Signaling
29 Eicosanoids as Mediators and Therapeutic Targets in Airway Inflammation
30 Nitric Oxide, Reactive Nitrogen Species, and Lipid Mediator Generation in the Lung
31 Adenosine Signaling and Lung Inflammation
32 Complement Factor 5 in Asthma
33 Complement Anaphylatoxin Receptors in Asthma
34 Inflammatory and Structural Changes in Airway Remodeling of Asthma
35 The Role of Proteases in Airway Remodeling
36 Airway Smooth Muscle
37 Cellular Trafficking of ß2-Adrenergic Receptors
38 Genetic Targets in Asthma
39 Application of Pharmacogenetics to the Therapeutics of Asthma
40 DNA-Based Immunotherapy for Asthma
41 Allergen Immunotherapy
42 Peptide-Based Therapy for Asthma
43 Reducing IgE Levels as a Strategy for the Treatment of Asthma
44 Strategies to Reduce Excessive Mucus Secretion in Airway Inflammation

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Tags: N Tony Eissa, David P Huston, Biology, Disease

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