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Liver-breast communication of adipocyte-orientedexosomes drives primary mammary cancerprogression by Chunni Li & Yiwen Lu & Yihong Li & Ting Liu & Hong Deng & Mingchao Gao & Boxuan Zhou & Jiayu Liu & Junchao Cai & Di Huang & Linbin Yang & Jin Jin & Dongming Kuang & Shicheng Su instant download

  • SKU: EBN-239206218
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Instant download (eBook) Liver-breast communication of adipocyte-orientedexosomes drives primary mammary cancerprogression after payment.
Authors:Chunni Li & Yiwen Lu & Yihong Li & Ting Liu & Hong Deng & Mingchao Gao & Boxuan Zhou & Jiayu Liu & Junchao Cai & Di Huang & Linbin Yang & Jin Jin & Dongming Kuang & Shicheng Su
Pages:updating ...
Year:2025
Publisher:x
Language:english
File Size:11.98 MB
Format:pdf
Categories: Ebooks

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Liver-breast communication of adipocyte-orientedexosomes drives primary mammary cancerprogression by Chunni Li & Yiwen Lu & Yihong Li & Ting Liu & Hong Deng & Mingchao Gao & Boxuan Zhou & Jiayu Liu & Junchao Cai & Di Huang & Linbin Yang & Jin Jin & Dongming Kuang & Shicheng Su instant download

Cell Metabolism, Corrected proof. doi:10.1016/j.cmet.2025.08.012

SUMMARYThe incidence of certain types of extrahepatic cancers significantly increases in nonalcoholic fatty liver disease (NAFLD), the mechanisms of which are elusive. Here, we demonstrate that NAFLD is correlated with ahigher risk of breast cancer in individuals with atypical hyperplasia and poor prognosis in patients with breastcancer. In mice, fatty liver exosomes are preferentially accumulated in adipocytes, and their enrichment inmammary adipocytes fosters a pro-tumor breast microenvironment. Adipocyte tropism is dictated by thebinding of exosomal ErbB4 to neuregulin 4 (Nrg4). tRNA methyltransferase 10 homolog C (TRMT10C) in fattyliver exosomes translocates to mitochondria and inhibits Nd5 and Nd6 mRNA translation by inducing N1-methyladenosine modifications in adipocytes. ND5 and ND6 reduction increases reactive oxygen speciesand consequently enhances free fatty acid release, which fuels tumor progression. Plasma ErbB4+ exosomesare an independent prognostic factor for patients with breast cancer and comorbid NAFLD. Collectively, wereveal a liver-breast metabolic remote interaction that drives cancer development.

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